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Bilingualism is a long-term cognitively challenging experience that modulates metabolite concentrations in the healthy brain
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In: Scientific Reports ; 11 (2021). - 7090. - Springer Nature. - eISSN 2045-2322 (2021)
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Bilingualism is a long-term cognitively challenging experience that modulates metabolite concentrations in the healthy brain
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In: Sci Rep (2021)
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What Have We Learned About Bilingualism? Regarding Nichols et al. (2020)
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Referee report. For: Early childhood bilingualism: effects on brain structure and function [version 1; peer review: 1 approved] ...
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The cognitive and neurological effects of bilingualism on healthy ageing and the progression of dementia: a longitudinal study ...
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The effect of bilingualism on brain development from early childhood to young adulthood
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In: Brain Struct Funct (2020)
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Online psycholinguistic methods in second language acquisition research
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Understanding structural plasticity in the bilingual brain: The Dynamic Restructuring Model
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Brain changes associated with language development and learning: A primer on methodology and applications
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Morphological processing in the brain : the good (inflection), the bad (derivation) and the ugly (compounding)
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In: Cortex ; 116 (2019). - S. 4-44. - ISSN 0010-9452. - eISSN 1973-8102 (2019)
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Redefining bilingualism as a spectrum of experiences that differentially affects brain structure and function
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Brain adaptations and neurological indices of processing in adult Second Language Acquisition: challenges for the Critical Period Hypothesis
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Abstract:
Stemming from the seminal work of Penfield and Roberts (1959) and Lenneberg (1967), a major question in adult language learning studies—indeed one that transcends all paradigms—has involved the extent to which adult language acquisition and processing is destined to be fundamentally different in adulthood compared to childhood. The basis of the original claims of the Critical Period Hypothesis (Lenneberg, 1967) regards neurological maturation after puberty; brain plasticity is said to be lost or greatly reduced, rendering the mechanisms that underlie language learning necessarily distinct and thus disadvantaging adults. No one denies that child and adult developmental paths differ; however, the evidence that is used to support critical/sensitive period effects are decisively not clear (see e.g., Abrahamsson and Hyltenstam 2009; Long, 2005, 2013; DeKeyser, 2000 as compared to Rothman, 2008; Bialystok and Hakuta 1994; Birdsong and Molis, 2001; Birdsong and Vanhove, 2016; Birdsong, 2014 for review and opposing views). With few exceptions, the vast majority of “relevant evidence” on the matter comes from behavioral experimentation or spontaneous production, most often from L2 populations not exposed to the target language in a way similar to child L1 acquirers (e.g., adults tend to be classroom learners and children tend to be naturalistic learners). In the past two decades, technologies have progressed that permit us to have a renewed look at the Critical Period debate. That the healthy brain remains plastic throughout the lifespan is no longer controversial within neurocognitive psychology (see Fuchs and Flügge, 2014 for review). And so, the neuro-maturational basis of the Critical Period Hypothesis advocated originally in Lenneberg (1967) and assumed by many ever since is necessarily challenged. In this chapter, we focus on how neurolinguistic evidence—EEG/ERP and (f)MRI data—can help us adjudicate between various views regarding the Critical Period debate and how to best account for the ubiquitously noted differences that align with age of acquisition effects in language acquisition/processing.
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URL: https://centaur.reading.ac.uk/76040/1/Chapter_Brain_adaptations_final.pdf https://centaur.reading.ac.uk/76040/
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Morphological processing in the brain: the good (inflection), the bad (derivation) and the ugly (compounding)
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